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Mammalian thioredoxin reductase: oxidation of the C-terminal cysteine/selenocysteine active site forms a thioselenide, and replacement of selenium with sulfur markedly reduces catalytic activity

it impairs SM microcirculation, lipid oxidation, mitochondrial activity, and glucose metabolism, which might aggravate metabolic dysfunction and IR

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Th17 responses and natural IgM antibodies are related to gut microbiota composition in systemic lupus erythematosus patients

Translating results from the nematode model to human disease further allows the assumption that both L-carnitine deficiency and accumulation of -BB play an independent but synergistic role in the pathogenesis of human BBOX1 deficiency